Mnemonic

Asthma Physiology

A memory aid for the physiological changes in acute asthma.

Expansion

Bronchoconstriction, gas trapping and hyperinflation with V/Q mismatch

Expansion

The sequence:

  1. Bronchoconstriction, mucosal oedema and mucus plugging raise airway resistance
  2. Expiratory flow limitation with dynamic compression traps gas
  3. Hyperinflation raises functional residual capacity; this splints airways open but forces breathing onto the flat, stiff part of the compliance curve
  4. Work of breathing rises sharply and the diaphragm is flattened and mechanically disadvantaged
  5. V/Q mismatch causes hypoxaemia

Carbon dioxide follows a characteristic course: low initially from hyperventilation, then normalising, then rising as the patient tires. A “normal” carbon dioxide in a distressed asthmatic is therefore a warning sign, not reassurance.

Other markers of severity: silent chest (too little airflow to generate wheeze), pulsus paradoxus from exaggerated inspiratory swings, inability to complete sentences, and exhaustion.

Auto-PEEP from incomplete exhalation impedes venous return and complicates mechanical ventilation, which requires long expiratory times and permissive hypercapnia.