Mnemonic

Blood-Brain Barrier

A memory aid for the structure and significance of the blood-brain barrier.

Expansion

Tight junctions between endothelial cells, supported by astrocyte foot processes

Expansion

Structure

  • Tight junctions between capillary endothelial cells, with no fenestrations
  • Thick basement membrane
  • Astrocyte foot processes inducing and maintaining the barrier
  • Pericytes
  • Efflux pumps such as P-glycoprotein actively removing drugs

Crosses freely: oxygen, carbon dioxide, water, and lipid-soluble drugs such as anaesthetics and many psychotropics. Glucose and amino acids need specific transporters.

Excluded: large or charged molecules, most antibiotics, catecholamines, and protein.

Circumventricular organs lack the barrier, because they must sample blood: the area postrema (chemoreceptor trigger zone), organum vasculosum (osmoreceptors), subfornical organ, median eminence and posterior pituitary.

Clinical consequences: levodopa crosses but dopamine does not, which is why the precursor is given with a peripheral decarboxylase inhibitor; meningitis disrupts the barrier, which is why penicillin becomes effective and why steroids are given to limit inflammation; and the barrier is immature in neonates, allowing kernicterus.