Mnemonic

Facial Nerve Palsy Localisation

A memory aid for distinguishing an upper from a lower motor neurone facial palsy.

Expansion

Forehead sparing separates the two

Expansion

The forehead has bilateral cortical representation, so an upper motor neurone lesion on one side is compensated by the other hemisphere.

Upper motor neurone Lower motor neurone
Forehead Spared, can raise eyebrows Involved, cannot raise eyebrow
Eye closure Preserved Weak, Bell’s phenomenon visible
Emotional movement Often preserved Lost
Common causes Stroke, tumour Bell’s palsy, Ramsay Hunt, parotid tumour

Localising a lower motor neurone palsy by the branches given off along the course

  • Above the geniculate ganglion: weakness plus reduced lacrimation, hyperacusis (stapedius) and loss of taste on the anterior two thirds of the tongue (chorda tympani)
  • Between the ganglion and the stapedial branch: weakness, hyperacusis and taste loss, lacrimation intact
  • Below the chorda tympani: weakness alone, which localises to the parotid or the distal nerve

Causes

  • Bell’s palsy, a diagnosis of exclusion, of abrupt onset, treated with prednisolone within 72 hours plus meticulous eye care with lubricants and taping
  • Ramsay Hunt syndrome: vesicles in the ear canal or palate, severe pain, often with vestibulocochlear involvement. Worse prognosis, and requires aciclovir plus steroid
  • Parotid malignancy, cholesteatoma, trauma, Lyme disease, sarcoidosis, and bilateral palsy, which suggests Guillain-Barré, Lyme, sarcoid or HIV

A slowly progressive facial palsy over weeks is a tumour until proven otherwise, since Bell’s palsy is sudden and begins to recover within three weeks.