Mnemonic

Infective Endocarditis Pathology

A memory aid for the organisms, valves and complications of endocarditis.

Expansion

Vegetations on damaged valves, with embolic and immune complications

Expansion

Organisms

  • Staphylococcus aureus: the commonest overall, acute, aggressive, and typical of intravenous drug users (affecting the tricuspid valve) and prosthetic valves
  • Streptococcus viridans: subacute, after dental procedures, on previously damaged valves
  • Streptococcus gallolyticus (bovis): associated with colorectal carcinoma, so colonoscopy is indicated
  • Staphylococcus epidermidis: prosthetic valves
  • HACEK organisms, and culture-negative causes such as Coxiella and Bartonella

Valves: mitral most often, then aortic; tricuspid in intravenous drug users because it is the first valve encountered by injected organisms.

Complications

  • Embolic: stroke, splenic and renal infarcts, mycotic aneurysm, Janeway lesions (painless palmar macules)
  • Immune complex: Osler nodes (painful finger pulp nodules), Roth spots, glomerulonephritis, splinter haemorrhages
  • Local: valve destruction with regurgitation, abscess, heart block

Diagnosis uses the modified Duke criteria, combining blood cultures and echocardiographic evidence with minor criteria.