Mnemonic

Intracranial Haemorrhage Types

A memory aid for the patterns of intracranial bleeding.

Expansion

Extradural, subdural, subarachnoid and intracerebral, each with a characteristic vessel and shape

Mnemonic

Four compartments, four appearances:

Type Shape on CT Vessel Typical patient
Extradural Biconvex, does not cross sutures Middle meningeal artery Young, temporal trauma, lucid interval
Subdural Crescent, crosses sutures Bridging veins Elderly, alcohol, anticoagulated, minor trauma
Subarachnoid Blood in the sulci and basal cisterns Berry aneurysm Thunderclap headache
Intracerebral Within the parenchyma Small perforators Hypertension, basal ganglia

“Extradural stops at sutures, subdural stops at the midline.”

Extradural is arterial so it deteriorates quickly, classically after a lucid interval; subdural is venous so it can be chronic and present with fluctuating confusion weeks later.

Subarachnoid haemorrhage requires CT within 6 hours (over 95 per cent sensitive), and if negative and clinically suspected, a lumbar puncture at 12 hours for xanthochromia.

Expansion

Type Vessel Shape on CT Typical setting
Extradural Middle meningeal artery Lens (biconvex), does not cross sutures Temporal skull fracture, young adult, lucid interval
Subdural Bridging veins Crescentic, crosses sutures Elderly, alcohol, anticoagulants; minor trauma
Subarachnoid Berry aneurysm rupture Blood in sulci and basal cisterns Thunderclap headache, meningism
Intracerebral Charcot-Bouchard microaneurysms Within parenchyma Hypertension, basal ganglia

The shape difference is anatomical: the dura is adherent at sutures, so extradural blood cannot cross them but is powerful enough to strip dura from bone. Subdural blood lies inside that attachment and spreads freely.

Subdural is common in the elderly and in alcohol dependence because cerebral atrophy stretches the bridging veins, so trivial trauma tears them, and the history of injury is often absent.

Subarachnoid haemorrhage risk factors: hypertension, smoking, polycystic kidney disease, Ehlers-Danlos and coarctation. Complications include rebleeding, vasospasm at days 3 to 14 (treated with nimodipine), hydrocephalus and hyponatraemia.