Mnemonic

Oedema Mechanisms in Pathology

A memory aid for the pathological classification of oedema.

Expansion

Transudate from pressure change; exudate from increased permeability

Expansion

Transudate Exudate
Mechanism Altered hydrostatic or oncotic pressure Increased permeability
Protein Low (under 30 g/l) High (over 30 g/l)
Specific gravity Under 1.012 Over 1.020
LDH Low High
Cells Few Many
Causes Heart failure, cirrhosis, nephrotic syndrome Infection, malignancy, infarction, inflammation

Light’s criteria identify a pleural exudate if any one holds: fluid to serum protein ratio above 0.5, fluid to serum LDH above 0.6, or fluid LDH above two-thirds the upper limit of normal serum LDH. They are deliberately weighted to avoid missing an exudate.

For ascites, the serum-ascites albumin gradient is more useful than the exudate and transudate distinction: a gradient of 11 g/l or more indicates portal hypertension, and below that suggests peritoneal disease such as malignancy, tuberculosis or pancreatitis.

Naming by site is worth keeping straight: effusion in a body cavity, ascites in the peritoneum, anasarca for generalised severe oedema, and hydrothorax, hydropericardium and hydrocephalus for the respective spaces.