Mnemonic

Primary Immunodeficiencies

A memory aid for classifying inherited immune defects by the arm affected.

Expansion

B cell, T cell, combined, phagocyte or complement

Mnemonic

Match the infection to the arm of the immune system that has failed:

  • B cell (antibody) - recurrent bacterial infections, especially encapsulated organisms and sinopulmonary infection, starting after 6 months as maternal IgG wanes. Bruton’s X-linked agammaglobulinaemia, common variable immunodeficiency, selective IgA deficiency (the commonest of all)
  • T cell - viral, fungal and opportunistic infections, failure to thrive from birth. DiGeorge syndrome (22q11: thymic aplasia, hypocalcaemia, cardiac defects)
  • Combined - SCID, presenting in infancy, requiring bone marrow transplantation
  • Phagocyte - abscesses and catalase positive organisms. Chronic granulomatous disease, diagnosed by the nitroblue tetrazolium or dihydrorhodamine test
  • Complement - early components: immune complex disease and lupus. Terminal components C5 to C9: recurrent Neisseria infections

“Bacteria means B cells, viruses and fungi mean T cells, abscesses mean phagocytes, Neisseria means complement.”

Live vaccines are contraindicated in T cell and combined deficiencies.

Expansion

Arm Example Typical infections
B cell (antibody) Bruton X-linked agammaglobulinaemia; selective IgA deficiency (commonest); common variable immunodeficiency Encapsulated bacteria, recurrent sinopulmonary infection, giardia
T cell DiGeorge (22q11 deletion, absent thymus) Viruses, fungi, Pneumocystis, mycobacteria
Combined SCID Everything; presents in infancy, fatal without transplant
Phagocyte Chronic granulomatous disease (NADPH oxidase), leucocyte adhesion deficiency Catalase-positive organisms: Staphylococcus, Aspergillus, Serratia
Complement C5 to C9 deficiency; C1 esterase inhibitor deficiency Neisseria; hereditary angioedema

Antibody deficiencies typically present after 6 months, once maternal IgG has waned, whereas severe T cell defects present earlier.

Warning signs prompting investigation: recurrent severe infections, unusual organisms, failure to thrive, family history, and infection with live vaccines.

Two specific associations worth remembering: DiGeorge with hypocalcaemia and cardiac outflow defects, and C1 esterase inhibitor deficiency causing angioedema that does not respond to adrenaline, steroids or antihistamines.