Mnemonic

Interpreting Raised Alkaline Phosphatase

A memory aid for finding the source of a raised ALP.

Expansion

Liver, bone, placenta or gut, distinguished by GGT and clinical context

Expansion

Hepatobiliary (GGT also raised)

  • Biliary obstruction: stones, tumour, stricture
  • Primary biliary cholangitis, primary sclerosing cholangitis
  • Drugs, infiltration, metastases

Bone (GGT normal)

  • Paget disease: characteristically a very high ALP with normal calcium and phosphate
  • Osteomalacia and rickets
  • Bone metastases, healing fracture, hyperparathyroidism
  • Normal growth in children and adolescents, where levels several times the adult range are expected

Physiological

  • Pregnancy, third trimester, from placental production

Other: ALP can be low in hypophosphatasia, malnutrition, hypothyroidism, Wilson disease and zinc deficiency.

If the source remains unclear, isoenzyme analysis separates the fractions directly.

A useful discipline is to interpret ALP alongside GGT, calcium, phosphate and the clinical picture rather than in isolation, since it is one of the most commonly abnormal results on routine testing and the great majority have a benign explanation.