Mnemonic

Renal Compensation for Acid-Base Disorders

A memory aid for the timing and extent of renal compensation.

Expansion

Slow but powerful, taking days but capable of large adjustments

Expansion

Respiratory compensation Renal compensation
Responds to Metabolic disorders Respiratory disorders
Speed Minutes to hours Days, 3 to 5 for completion
Mechanism Change in alveolar ventilation Bicarbonate handling and acid excretion
Capacity Limited Large

Renal mechanisms in acidosis:

  • Increased hydrogen ion secretion and bicarbonate reabsorption
  • Greatly increased ammoniagenesis from glutamine, which can rise several-fold over days and is the main adaptive component
  • Increased titratable acid excretion

In alkalosis: bicarbonate is excreted, giving an alkaline urine.

Neither system fully corrects the pH, since complete correction would remove the stimulus for compensation. A pH that has returned entirely to normal therefore suggests either a mixed disorder or a chronic, fully compensated respiratory state.

One important exception: in hypovolaemia, the kidney prioritises volume over pH and retains bicarbonate along with sodium, which is how a contraction alkalosis is maintained despite the alkalaemia.