Mnemonic

Upper Versus Lower Motor Neurone Lesions

A memory aid for distinguishing upper from lower motor neurone signs.

Expansion

Upper gives spasticity and brisk reflexes; lower gives wasting and absent reflexes

Mnemonic

“Everything goes up in upper, everything goes down in lower”:

Upper motor neurone Lower motor neurone
Tone Increased, spastic Reduced, flaccid
Reflexes Brisk, clonus Absent or reduced
Plantars Extensor (Babinski) Flexor
Wasting Late, disuse only Early and marked
Fasciculation Absent Present
Pattern of weakness Pyramidal: extensors weak in the arm, flexors weak in the leg Follows the nerve or root

“Fasciculation is the giveaway for lower motor neurone.”

The pyramidal pattern is worth knowing precisely: in the arm the extensors are weaker, in the leg the flexors are weaker, which is what produces the flexed arm and extended leg of a hemiplegic posture.

Motor neurone disease is the classic combination of both, giving wasting and fasciculation alongside brisk reflexes and extensor plantars, with no sensory signs.

Expansion

Feature Upper motor neurone Lower motor neurone
Tone Increased (spastic) Decreased (flaccid)
Reflexes Brisk, with clonus Absent or reduced
Plantar response Extensor (Babinski) Flexor
Wasting Minimal (disuse only) Marked
Fasciculation Absent Present
Pattern of weakness Pyramidal: extensors weak in the arm, flexors weak in the leg Follows the nerve or root

Spasticity is velocity dependent and shows the clasp-knife phenomenon, in contrast to the lead-pipe or cogwheel rigidity of extrapyramidal disease.

An important caveat: in an acute upper motor neurone lesion such as stroke or spinal cord injury, there is an initial period of flaccidity and areflexia (spinal shock) lasting days to weeks before spasticity develops.

Motor neurone disease is the classic condition combining both, with wasting and fasciculation alongside brisk reflexes.