Mnemonic

Pain Pathways

A memory aid for the transmission of pain from periphery to cortex.

Expansion

Transduction, transmission, modulation and perception

Expansion

  1. Transduction: noxious stimuli activate nociceptors. Targets: NSAIDs and local anaesthetics
  2. Transmission: A delta fibres carry fast sharp pain, C fibres slow dull pain, to the dorsal horn and then the spinothalamic tract. Targets: local anaesthetics, regional blocks
  3. Modulation: in the dorsal horn, by the gate mechanism and by descending inhibition from the periaqueductal grey using endogenous opioids, serotonin and noradrenaline. Targets: opioids, tricyclics, SNRIs, clonidine
  4. Perception: cortical and limbic processing, influenced by attention, mood and expectation. Targets: psychological therapies, and the placebo response

Types of pain

  • Nociceptive: tissue injury, responds well to conventional analgesia
  • Neuropathic: nerve damage, with burning, shooting pain, allodynia and hyperalgesia. Responds poorly to opioids and better to gabapentinoids, tricyclics and SNRIs
  • Nociplastic: altered central processing, as in fibromyalgia

Wind-up and central sensitisation explain why untreated acute pain becomes harder to control, and why pre-emptive and multimodal analgesia is preferred.