Mnemonic

Rhabdomyolysis Physiology

A memory aid for the consequences of muscle breakdown.

Expansion

Myoglobin, potassium, phosphate and creatine kinase are released into the circulation

Expansion

Released from damaged muscle

  • Myoglobin: nephrotoxic, causing acute kidney injury by tubular obstruction, direct toxicity and renal vasoconstriction
  • Potassium: hyperkalaemia, often severe and early
  • Phosphate: hyperphosphataemia, causing hypocalcaemia by precipitation
  • Creatine kinase: the diagnostic marker, typically above 5 times normal and often in the tens of thousands
  • Urate and lactate

Later, calcium is released back from damaged muscle, causing rebound hypercalcaemia during recovery, which is why calcium should be given cautiously.

Causes: crush injury, prolonged immobility, seizures, extreme exertion, malignant hyperthermia, neuroleptic malignant syndrome, statins, alcohol, and compartment syndrome.

Management

  • Aggressive intravenous fluid, the mainstay, aiming for a good urine output
  • Treat hyperkalaemia
  • Monitor for compartment syndrome, which can be both cause and consequence
  • Urinary alkalinisation is of debated benefit